An inflammation of the vagina called vaginitis can cause pain, itching, and discharge. The cause is typically an infection or an imbalance in the vaginal bacterial balance. Other causes of vaginitis include certain skin conditions and decreased estrogen levels following menopause. One of the most prevalent forms of vaginitis is bacterial vaginosis. This happens when the natural equilibrium is upset by an overabundance of the germs that are naturally present in your vagina. as well as fungal infection. Usually, a naturally occurring fungus known as Candida albicans is the source of these. Urinary tract infections may occur with vaginitis. 64% of those involved in this study had a urinary tract infection correlated with vaginitis. The type of vaginitis in adult women was identified through investigation. 56% of the specimens were vaginal fungal infections, 22% were bacterial infections, and the remaining percentage were a combination of bacterial and fungal infections. This is where the challenge in treating such instances resides.

Guillain-Barré syndrome (GBS) is an immune-mediated polyradiculopathy that typically presents with neurological symptoms within four weeks of a preceding infection. It is characterized by symmetrical, ascending muscle weakness and areflexia, often sparing cranial nerves. Two therapeutic modalities are currently established—intravenous immunoglobulin (IVIG) and plasmapheresis—with comparable efficacy. Despite treatment, a subset of patients may experience severe functional impairment or even mortality. We present the case of a patient with GBS who showed poor response to IVIG, developed respiratory failure, and underwent prolonged hospitalization with multiple complications. A multidisciplinary team enabled home discharge with mechanical life-support ventilation, facilitating continued rehabilitation in a safer, non-hospital environment.

The aim of our study is to collect epidemiological, clinical, therapeutic, and evolutionary data on hand burns in order to evaluate our care protocols, propose preventive measures, and develop recommendations tailored to our context. Materials and methods: This is a retrospective descriptive and analytical study spanning four years, from January 2017 to December 2020, within the plastic, reconstructive, and burn surgery department at the Mohamed V Military Teaching Hospital in Rabat. During this period, we collected data on 102 patients with burns to at least one hand who were admitted to our department for treatment of burns in the acute phase. Results: This study shows that the most common epidemiological profile is that of a male patient, aged between 35 and 45, admitted for superficial or intermediate second-degree burns affecting the dorsal surface of both hands and the face, caused by a domestic accident involving the explosion of a 3 kg gas cylinder or a pressure cooker. Discussion: Treating a burned hand is a difficult challenge for the surgeon. However, it is a situation that surgeons often face due to the high number of hand burns. The diagnosis of depth, quality, and early initial treatment are therefore essential to reduce functional and aesthetic complications. Conclusion: Early, adequate, multidisciplinary care remains the cornerstone for limiting the sequelae of hand burns and offering the patient an acceptable quality of life.

Bullous pemphigoid (BP) represents one of the most prevalent forms of bullous diseases. Nevertheless, its localized variant, known as Localized Bullous Pemphigoid (LBP), is relatively uncommon, comprising only 2.5% of all BP cases. LBP is typically associated with trauma and may manifest on any wound or surgical site. Although chronic venous stasis has not been well-documented as a predisposing factor. We present the case of an 80-year-old man who exhibited blistering lesions localized to his left lower limb. He had a history of chronic venous insufficiency (CIV) on both legs, with no improvement after surgery. Clinical-pathology correlation concluded with the diagnosis of LBP. Treatment with clobetasol topical ointment and compression therapy led to complete remission of the skin lesions after one month. This report underscores chronic venous stasis as a contributing trigger for LBP.

The evolution of knowledge regarding the anatomy of the intestine throughout Greece-Arabic period and the Modern period is described. The common opinions of this organ at various points throughout this timeline are shown. The original words of great physicians from the period of time stretching from Ancient Egypt to the Avicenna era and hakim Kābbiruddin are quoted and discussed to demonstrate how knowledge of the intestine has evolved. Unani Medicine is a medical system that treats with the management of health and disease. Jalinus (Galen, 200 AD) observed that Small intestine heal more quickly and with lesser complications than wounds of the large intestine because of good blood supply. Avicenna stated that duodenal mucosa lacked the circular folds of mucosa (plicae circulares) found in the jejunum and ileum. According to Avicenna, the jejunum has a abundant blood vessels. It consisting of large, tall and closely packed circular folds of mucosa than the ileum.

This study addressed the importance of bacterial infections in diabetic foot as a serious complication that can lead to ulcers and chronic inflammation, increasing the risk of limb loss. The study reviewed the most common types of bacteria that infect diabetic foot and the role of these bacteria in complicating wounds and injuries. Thirty foot swab samples (15 patients and 15 controls) were collected and cultured in the microbiology unit at Al-Marjan Hospital in Babylon, statistical analysis was done and results shows some types of bacteria are more common, such as Staphylococcus aureus. also noted that men with diabetic foot ulcer more than women, and that middle-aged people (aged 52-62, 63-73) are more likely to be infected with diabetic foot. This study concluded that staphylococcus aureus was common pathogenic bacteria that cause diabetic foot ulcer, also male sex is regarded as risk factor for diabetic ulcer. Additionally no relation between agent species with severity of disease.

Background: Rheumatoid arthritis (RA) is a chronic autoimmune disease characterized by inflammation, joint destruction, and immune dysregulation. Vitamin D, a key regulator of the immune system, has been implicated in various autoimmune diseases, including RA. However, the exact role of vitamin D in immune modulation and its potential relationship with immune markers in RA patients remains underexplored. Objective: This study aimed to investigate the relationship between vitamin D deficiency and immune dysregulation in RA patients, with a focus on inflammatory markers, cytokines, and regulatory T-cells. Methodology: A cross-sectional study was conducted from 1st January to 30th August 2024, including 100 RA patients (50 males, 50 females) and 50 control participants (25 males, 25 females). Blood samples were collected from all participants to measure serum vitamin D levels, inflammatory markers (C-reactive protein [CRP], erythrocyte sedimentation rate [ESR]), and immune parameters (IL-10, IL-17, and regulatory T-cells). Data were analyzed using correlation analysis to examine relationships between vitamin D levels and immune parameters. Results: RA sufferers exhibited drastically decrease nutrition D degrees as compared to controls (18.5 ± 6.2 ng/mL vs. 32.1 ± 7.8 ng/mL, p < 0.001). Additionally, better inflammatory markers had been determined in RA patients, with CRP and ESR levels considerably expanded compared to controls (p < 0.001). A widespread high-quality correlation turned into found among vitamin D stages and IL-10 (r = 0.582, p < 0.001) and regulatory T-cells (r = 0.421, p < 0.001), whilst an inverse correlation changed into determined with IL-17 (r = -0.645, p < 0.001). Moreover, a terrible correlation between vitamin D stages and ESR (r = -0.512, p < 0.001) become additionally cited, suggesting a link between nutrition D deficiency and systemic irritation in RA. Conclusion: The outcomes of this observe assist the position of diet D in immune modulation and irritation in RA patients. Vitamin D deficiency turned into associated with better inflammatory markers and adjusted immune responses, together with accelerated levels of seasoned-inflammatory cytokines (IL-17) and reduced regulatory T-cellular characteristic. These findings highlight the potential therapeutic function of vitamin D supplementation in managing immune dysregulation and infection in RA. Further studies are had to affirm these outcomes and discover the medical benefits of diet D in RA remedy.